artículo académico

• Omp25‐dependent engagement of SLAMF1 by Brucella abortus in dendritic cells limits acute inflammation and favours bacterial persistence in vivo.  Cellular Microbiology.  22:e13164. 2020
• Infection by Brucella melitensis or Brucella papionis modifies essential physiological functions of human trophoblasts.  Cellular Microbiology.  21:e13019. 2019
• Cyclic β-Glucans at the bacteria-host cells interphase: one sugar ring to rule them all.  Cellular Microbiology.  20:e12850. 2018
• Identification of lptA, lpxE, and lpxO, Three Genes Involved in the Remodeling of Brucella Cell Envelope.  Frontiers in Microbiology.  8. 2018
• Persistence of Brucella abortus in the Bone Marrow of Infected Mice.  Journal of Immunology Research.  2018:1-8. 2018
• Immunomodulatory properties of Brucella melitensis lipopolysaccharide determinants on mouse dendritic cells in vitro and in vivo.  Virulence.  9:465-479. 2017
• Vaccine development targeting lipopolysaccharide structure modification.  Jiménez Osornio, Juan.  20:455-460. 2017
• Structural Studies of Lipopolysaccharide Defective Mutants from Brucella melitensis Identify a Core Oligosaccharide Critical in Virulence.  Journal of Biological Chemistry.  291:7727-7741. 2016
• Brucella abortus induces the premature death of human neutrophils through the action of its lipopolysaccharide.  PLos Pathogens.  11:e1004853. 2015
• Cervical Lymph Nodes as a Selective Niche for Brucella during Oral Infections.  PLoS ONE.  1-26. 2015
• Plasticity versus specificity in RTK signalling modalities for distinct biological outcomes in motor neurons.  BMC Biology.  12:56. 2014
• The identification of wadB, a new glycosyltransferase gene, confirms the branched structure and the role in virulence of the lipopolysaccharide core of Brucella abortus.  Microbial Pathogenesis.  73:53-59. 2014
• Toxicity of Bothrops sp snake venoms from Ecuador and preclinical assessment of the neutralizing efficacy of a polyspecific antivenom from Costa Rica.  Toxicon.  88:34-37. 2014
• Estrada, Jose.  PLos Pathogens.  9:e1003167. 2013
• In Vivo Identification and Characterization of CD4(+) Cytotoxic T Cells Induced by Virulent Brucella abortus Infection.  PLoS ONE.  8:e82508. 2013
• Lipopolysaccharide as a target for brucellosis vaccine design.  Microbial Pathogenesis.  58:29-34. 2013
• Pathogenic Brucellae Replicate in Human Trophoblasts.  Journal of Infectious Diseases.  207:1-9. 2013
• The Lipopolysaccharide Core of Brucella abortus Acts as a Shield Against Innate Immunity Recognition.  PLos Pathogens.  8:e1002675. 2012
• Brucella abortus Ornithine Lipids Are Dispensable Outer Membrane Components Devoid of a Marked Pathogen-Associated Molecular Pattern.  PLoS ONE.  6:e16030. 2011
• Evolution and taxonomic classification of human papillomavirus 16 (HPV16)-related variant genomes.  PLoS ONE.  6:1-16. 2011
• Pool-Specific Regulation of Motor Neuron Survival by Neurotrophic Support.  The Journal of Neuroscience.  31:11144-11159. 2011
• The Glyceraldehyde-3-Phosphate Dehydrogenase and the Small GTPase Rab 2 Are Crucial for Brucella Replication.  PLos Pathogens.  5:e100487. 2009
• Brucellosis Vaccines: Assessment of Brucella melitensis Lipopolysaccharide Rough Mutants Defective in Core and O-Polysaccharide Synthesis and Export.  PLoS ONE.  3:e2760. 2008
• Cardiotrophin-1 requires LIFRbeta to promote survival of mouse motoneurons purified by a novel technique.  The Journal of Neuroscience.  55:119-126. 1999
• Role of neurotrophic factors in motoneuron development.  Journal of Physiology-Paris.  92:279-281. 1998
• Synergistic Effects of Schwann- and Muscle-Derived Factors on Motoneuron Survival Involve GDNF and Cardiotrophin-1 (CT-1).  The Journal of Neuroscience.  18:1440-1448. 1998
• Hepatocyte growth factor (HGF/SF) is a muscle-derived survival factor for a subpopulation of embryonic motoneurons.  Development.  124:2903-2913. 1997
• Cardiotrophin-1, a Cytokine Present in Embryonic Muscle, Supports Long-Term Survival of Spinal Motoneurons.  Neuron.  17:63-74. 1996
• Use of Chimeric F3-NCAM Molecules to Explore the Properties of VASE Exon in Modulating Polysialylation and Neurite Outgrowth.  Journal Cell Adhesion and Communication.  3:541-554. 1996
• Changes in myofibrillar components after skeletal muscle necrosis induced by a myotoxin isolated from the venom of the snake Bothrops asper.  Blackburn, Nicholas B..  52:25-36. 1990