artículo académico

• Identification of residues critical for toxicity in Clostridium perfringens phospholipase C, the key toxin in gas gangrene.  European Journal of Biochemistry.  267:5191-5197. 2017
• Reactive Oxygen Species and the MEK/ERK Pathway Are Involved in the Toxicity of Clostridium perfringens -Toxin, a Prototype Bacterial Phospholipase C.  Journal of Infectious Diseases.  206:1218-1226. 2012
• Comparison of a nontoxic variant of Clostridium perfringens α-toxin with the toxic wild-type strain.  Acta Crystallographica Section D: Biological Crystallography.  66:1067-1074. 2010
• A cellular deficiency of gangliosides causes hypersensitivity to Clostridium perfringens phospholipase C.  Journal of Biological Chemistry.  280:26680-26689. 2005
• Effects of Clostridium perfringens phospholipase C in mammalian cells.  Anaerobe.  10:115-123. 2004
• Tyrosine 331 and phenylalanine 334 in Clostridium perfringens α-toxin are essential for cytotoxic activity.  FEBS Letters.  495:172-177. 2001
• Identification of Residues in the Carboxy-Terminal Domain of Clostridium perfringens α-Toxin (Phospholipase C) Which Are Required for Its Biological Activities.  Archives of Biochemistry and Biophysics.  384:24-30. 2000
• UDP-glucose Deficiency Causes Hypersensitivity to the Cytotoxic Effect of Clostridium perfringens Phospholipase C.  Journal of Biological Chemistry.  273:24433-24438. 1998