Lugar de publicación
- Structural Studies of Lipopolysaccharide Defective Mutants from Brucella melitensis Identify a Core Oligosaccharide Critical in Virulence. 291:7727-7741. 2016
- Pseudomonas putida KT2440 strain metabolizes glucose through a cycle formed by enzymes of the Entner-Doudoroff, embden-meyerhof-parnas, and pentose phosphate pathways. 290:25920-25932. 2015
- Fructose 1-phosphate is the preferred effector of the metabolic regulator Cra of Pseudomonas putida. 286:9351-9359. 2011
- A cellular deficiency of gangliosides causes hypersensitivity to Clostridium perfringens phospholipase C. 280:26680-26689. 2005
- A Cellular UDP-glucose Deficiency Causes Overexpression of Glucose/Oxygen-regulated Proteins Independent of the Endoplasmic Reticulum Stress Elements. 279:21724-21731. 2004
- R-Ras Glucosylation and Transient RhoA Activation Determine the Cytopathic Effect Produced by Toxin B Variants from Toxin A-negative Strains of Clostridium difficile. 278:7956-7963. 2003
- Cysteine 38 in p65/NF-kB Plays a Crucial Role in DNA Binding Inhibition by Sesquiterpene Lactones. 276:13-20. 2001
- GTPases of the Rho subfamily are required for Brucella abortus internalization in nonprofessional phagocytes: Direct activation of Cdc42. 276:44435-44443. 2001
- In Vivo Proteolytic Degradation of the Escherichia coli Acyltransferase HlyC. 276:16660-16666. 2001
- UDP-glucose Deficiency Causes Hypersensitivity to the Cytotoxic Effect of Clostridium perfringens Phospholipase C. 273:24433-24438. 1998
- Cellular UDP-Glucose Deficiency Caused by a Single Point Mutation in the UDP-Glucose Pyrophosphorylase Gene. 272:23784-23791. 1997
- Neutralizing interaction between heparins and myotoxin II, A lysine 49 phospholipase A2 from Bothrops asper snake venom. Identification of A heparin-binding and cytolytic toxin region by the use of synthetic peptides and molecular modeling. 269:29867-29873. 1994
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Neutralizing interaction between heparins and myotoxin II, a lysine 49 phospholipase A
2 from Bothrops asper snake venom: Identification of a heparin-binding and cytolytic toxin region by the use of synthetic peptides and molecular modeling. 269:29867-29873. 1994